![]() ![]() Although all factors were evaluated as prognostic factors for functional outcome after surgical treatment of recent-onset macula-off RRD, epiretinal membrane formation was found as the only factor affecting postoperative visual acuity. The mean preoperative and postoperative logMAR BCVA were 1.1 ± 0.5 (20/250) and 0.14 ± 0.1 (20/30), respectively, after a mean of 16.7 ± 7.2 months. Correlation between preoperative and postoperative variables such as demographic and clinical data, SD-OCT findings and surgical factors, and postoperative functional outcomes were assessed.Įighteen women and 26 men, whose mean age at the onset of RRD was 51.7 ± 14.4 years, were evaluated. visual acuity (BCVA) and retinal and choroidal changes on spectral-domain optical coherence tomography (SD-OCT) were evaluated before and after surgery. To assess correlation between preoperative and postoperative findings and surgical factors, and postoperative functional outcomes after successful repair of acute macula-off rhegmatogenous retinal detachment (RRD).įorty-four eyes of 44 patients with recent-onset macula-off RRD who had primary pars plana vitrectomy were included in this retrospective study. Because of a high risk of vitreous incarceration within the tube, a complete pars plana vitrectomy should be performed prior to GDD implantation. However, GDD implantation should only be performed in carefully selected patients. Glaucoma progression is a major cause of visual decline post-KPro. Vitreoretinal, glaucoma and infectious complications caused BCVA loss in 16 eyes (21%) with KPro alone (p=0.13). Vitreous incarceration was the most common cause of tube occlusion. Seven eyes (39%) with KPro and GDD experienced vision loss due to complications such as glaucoma progression (three eyes, 22%), tube occlusion (four eyes, 22%) and choroidal haemorrhage (three eyes, 17%). Median BCVA at postoperative 6 months was 20/150 in both groups. KPro surgery was performed in 96 eyes: 18 eyes (19%) had KPro and GDD while 78 eyes (81%) had KPro only. BCVA and complications were tabulated and analysed. Retrospective case series of all patients who underwent KPro surgery at the Centre Hospitalier de l'Université de Montréal between 20. To compare the complications leading to best-corrected visual acuity (BCVA) loss in patients with Boston keratoprosthesis type 1 (KPro) and glaucoma drainage device (GDD) and those with KPro alone. Vitreoretinal surgeons should avoid warming intraocular infusion fluids to levels above room temperature. While retinal and retinal pigment epithelium damage was present after the body temperature exposure, no damage was detected after the room temperature exposure. A 35-minute exposure to body temperature fluid was compared with the same exposure during infusion of room temperature fluid. Cooling the infusion fluid from body to room temperature extended the damage threshold from approximately 25 to 60 minutes. ![]() Damage was determined ophthalmoscopically and histologically. Following vitrectomy, we exposed the retina of rabbits to light from an intraocular fiberoptic probe during infusion of fluid at body temperature (39 C) and compared this with exposures during infusion of room temperature fluid (22 C). ![]() We examined the ability of hypothermic infusion fluid to reduce the risk of light damage to the retina from the intraocular fiberoptic probe during vitreous surgery. Furthermore, no phototoxicity from the sustained retinal exposure to a fixed, implantable light source for a clinically relevant duration could be demonstrated. We have confirmed that a standard endoilluminator probe can create a phototoxic lesion in the experimental setting. A control eye treated with a 20-minute exposure to a standard endoiluminator probe at maximal intensity held 2 mm from the retinal surface demonstrated typical light and electron microscopic phototoxic abnormalities, whereas a retina similarly treated with a 5-minute exposure was normal. Light and electron microscopic examination failed to reveal any of the characteristic changes of retinal phototoxicity. No clinically apparent lesion developed in any of the experimentally treated eyes by 48 to 72 hours posttreatment. To assess for the possible development of a phototoxic lesion, we exposed five Dutch belted rabbit retinas to constant illumination with an implantable "chandelier" light source at maximal intensity for 1 hour.
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